Interruptions in the work of the heart: skipping strokes, fading, what to do

In this article: why the heart often beats with interruptions and there are missed strokes, acceleration and slowing of the rhythm, irregular cuts. Symptomatology of various disorders, how to establish a diagnosis and what to do if there is a pathology in the normal functioning of the heart.

Contents of the article:

  • Tachycardia
  • Extrasystoles
  • Bradycardia
  • Diagnosis of the heart in its failure
  • Prognosis for these pathologies

Cardiac contraction in a constant rhythm is the guarantee of normal functioning of all organs. The rhythm of the contractions corresponds to the accumulations of pacemaker cells( rhythm drivers) creating an electric pulse of constant frequency( sinus and atrioventricular nodes).The excitation wave that has arisen in this zone, along the conducting paths( the bundle of the Hyis) extends to all parts of the heart, causing a contraction.

Cardiac irregularities or arrhythmias are changes in the frequency of strokes associated with a disruption in the formation or excitation wave.

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The normal rhythm of heart contractions is 60-90 per minute, each stroke at an equal time. The source of the electric pulse is the sinus node.

Three major disorders:

  1. Tachycardia - acceleration of the heartbeat.
  2. Bradycardia is a slowing down of the heart muscle.
  3. Extrasystoles - additional abbreviations against the background of a normal rhythm.

Rhythm disorders can occur intermittently( paroxysm), be asymptomatic or quickly lead to poor health and cause death. Arrhythmias that are not associated with cardiac disease are well treated, and chronic diseases with myocardial damage require constant therapy.

Changes in the rhythmic work of the heart can not be missed, it is an occasion to consult a doctor to avoid arrhythmia. Patients with this pathology are treated by therapists and cardiologists, and in specialized centers - by arrhythmologists.

Tachycardia

Atrial and / or ventricular contraction with a frequency of more than 100 per minute.

Types of tachycardia

Types of tachycardia
Tachycardia

Atrial

Ventricular

Atrioventricular

Acute( first seen with a sharp period of any disease)

Paroxysmal( intermittent disturbance, there are periods of a normal pulse or there was only one episode of rhythmic disturbance)

Recurrentrepetitive heart failure )

Constant

By the mechanism of occurrence of Recurrent( reciprocal)

Automatic( ectopic)

Alopecia( trigger)

more detailed description of mechanism, see the section "Mechanism of Development" continued.

Mechanism of development of

Reciprocal( recurrent) mechanism of development accounts for 80% of all tachyarrhythmias. Due to disturbances in the motion of the excitation wave, the electric pulse moves in a circle, again exciting the same region of the heart.

Focal( automatic) tachycardia account for 10%, associated with impaired metabolism, due to increased excitability of the conductive pathways. Under such conditions, all impulses cause a contraction. Normally, for the emergence of an excitation wave, electrical activity of a certain level is necessary.

Triggered( triggered) tachyarrhythmias are caused by the appearance of an electric pulse exceeding the normal level: after the excitation wave is carried out, the residual pulse again triggers cardiac contraction.

Reasons for

Specific causes of Specific causes of
Heart muscle damage Blood supply disorder( infarction, chronic ischemia)

Muscular degeneration in connective( myopathy)

Chambers expansion or thickening( hypertrophy, pulmonary heart)

Inflammation( myocarditis)

Heart defects

Neoplasms withheart damage

Infringement of internal metabolism Decrease in potassium, magnesium level

Intoxication( alcohol, coffee, nicotine)

Kidney function deficiency

Medicinalth action Glycosides

Preparations against arrhythmias

stimulators of the sympathetic

Nervous System Oxygen starvation Anemia

lung diseases

Insufficient circulation

Endocrine diseases Hyperthyroidism

Diabetes

Other causes Injury

Syndromes WPW and braditahikardii

Causeless

Symptoms tachycardia

  • Heartbeats with interruptions.
  • Skipping strokes or increased heart beat.
  • Uneven, rapid heart rate.
  • Rapid fatigue with normal loads.
  • Rapid breathing, a feeling of lack of air with physical exertion.
  • Dizziness, darkening in the eyes, fainting.
  • Unsustainable blood pressure( abbreviated AD) with a tendency to decrease.
  • Pain in the heart.

With the development of severe cardiovascular complications( cardiac shock, pulmonary edema), the symptoms of cardiovascular failure are associated with a risk of cardiac arrest.

Tachycardia is acute or chronic, progressive in nature.

Overview of some types of tachycardia

Atrial fibrillation

Frequent, irregular atrial contraction. Is 75-85% of all supraventricular forms, occurs in 1-2% of the adult population. Rarely diagnosed before the age of 25, affecting people of senile and old age.

The disease is characterized by a high risk of death, the development of cardiac insufficiency, the formation of thrombi with their migration to the vessels of the lungs and brain. Continuously flowing asymptomatically, and after the appearance of the clinic quickly reduces the quality of life due to the inability to transfer even light loads.

Atrial fibrillation forms:

Atrial fibrillation forms:
Paroxysmal Variable arrhythmia

Self-sustained recovery of rhythm for 2-7 days

Persistent Requires medical intervention to restore sinus rhythm

Duration of episode more than 7 days

Persistent Pacing for 1 year when there isthe possibility of surgical treatment
Constant Absence of the effect of therapy

Patient's refusal from treatment

Atrial flutter

Accelerated, regular atrial contraction. Men are more often ill. Up to 50 years, the occurrence of 5 cases per 100 thousand population, with increasing age, the frequency of occurrence is growing exponentially.

It has a paroxysmal character, extremely rare is the chronic form. In the absence of treatment, 20% of the sick suddenly die.

In 20-35% of cases it is combined with atrial fibrillation. The clinical picture of a similar nature, but flutter quickly leads to heart failure with edematous syndrome.

Ventricular tachycardia

The source of the pathological excitation pulse is located in the ventricular myocardium or the terminal branches of the conducting system. The cause is only cardiac factors( 70-80% ischemic disease).

The lack of treatment leads to ventricular fibrillation and cardiac arrest.

Rare forms of

  • atrioventricular( nodular and focal);
  • sinus and atrial.

occur in 1-10% of tachyarrhythmias. The focal form is more often diagnosed in children, when the heart beats with a frequency of up to 210 per minute.

Interruptions in the heart are paroxysmal, rarely change into permanent form. They respond well to treatment.

Treatment of

Two approaches to therapy:

  1. Restore normal rhythm and maintain it.
  2. Monitor the level of tachycardia without restoring the rhythm.

The choice is individual. Regardless of what needs to be done, there are general rules:

  • to restore the rhythm in young people without disturbances of cardiac function;
  • maintain a heart rate of up to 90 per minute at rest and not above 115 with the load in elderly people with cardiac pathology;
  • use anticoagulants( warfarin, low molecular weight heparins) - prevent the formation of thrombi in the heart chambers;
  • to treat the causes of tachyarrhythmia.

Embodiments recovery rate

method Specific action agents
Cardioversion - "restart» heart Drug( Propafenol, Flecainide)

Electric( discharge current)

Hybrid

Medication Beta blockers

Calcium channel blockers

Glycosides

ablation - the destructionfoci with pathological electrical activity Catheter( endovascular, using burning or freezing)

Open heart surgery( used in casesthe need for an operation on other occasions)

Installation of pacemakers Installing a pacemaker or cardioverter defibrillator

The choice of tactics depends on the symptoms and degree of circulatory disturbance. Severe cardiopulmonary failure is an indication for an emergency electrical cardioversion. In non-threatening conditions, drug therapy is used, followed by a decision to perform ablation.

Control of the level of tachycardia

For this use the same antiarrhythmic drugs as for restoring the rhythm. A combination of several drugs is often required.

The effectiveness of treatment is determined by the causes. Without a primary pathology of the heart or its secondary change against a background of a prolonged absence of a sinus rhythm, a complete cure is possible. In tachycardia due to chronic diseases, antiarrhythmic therapy is lifelong.

Extrasystoles

Extrasystoles are an extraordinary reduction of the heart muscle.

Its forms:

Its localization: Sinus

Atrioventricular

Atrial

Ventricular

At frequency per hour Rare( & lt; 10)

Average frequency( 10-30)

Frequent( & gt; 30)

Density Single

Paired

Extrasystoles occur for the same reasons as tachycardia. Often provoke the onset of paroxysm of tachyarrhythmia.

Extrasystolia in most cases is asymptomatic, sometimes there are:

  • a heart failure;
  • sensation of an intensified heart beat or stop, heart sinking.

In heart failure, the course of the disease worsens, and the risk of death increases. Single extrasystoles occur in healthy people, do not require correction. Respond well to treatment.

Treatment of extrasystole associated with cardiac muscle damage includes:

  • beta blockers;
  • ablation of foci of high-frequency extrasystole.

Bradycardia

Bradycardia is the contraction of the atria and / or ventricles with a frequency of less than 60 per minute. Medical intervention requires bradyarrhythmia of the ventricles.

types type

sinus syndrome syndrome Atrioventricular blockades( in simple language - blockade of cardiac pulses in the heart)

paroxysmal

Chronic

Sinus node weakness

Decrease or turn off electrical activity of the main pacemaker -sinus node. The disease affects people of elderly and senile age, is rarely diagnosed in young people. Occurrence - 3 cases per 5000 population.

In the early stages without a decrease in the rhythm of less than 40 per minute, there are no symptoms. Distinguishes progressing course. Treatment improves overall well-being, but does not always affect life expectancy.

Atroventricular blockade of

There is a disturbance in the excitation wave along the conducting pathways to the ventricles. The occurrence is 2-5%.Progressing current.

There are 3 degrees of severity as the excitation wave is disturbed. Blockades of 2-3 degrees are distinguished by a high risk of cardiac arrest.

Causes of

  1. Cardiac factors, as with tachyarrhythmias.
  2. Increase in the level of potassium, magnesium.
  3. Diseases of the esophagus, stomach.
  4. Side effect of antiarrhythmic drugs, analgesics of the morphine group.
  5. Vagotonia( predominance of the action of the parasympathetic nervous system).
  6. Low level of thyroid function.
  7. Causeless.

Symptoms of

  • Reduced heart rate.
  • Interruptions in the heart, a sense of stopping it.
  • Expressed, growing weakness, inability to perform ordinary business.
  • Unstable blood pressure level, often - episodes of an increase with a low effect of treatment.
  • Impaired consciousness to deep fainting.
  • Chest pain without physical stress.

Treatment of

What to do in this pathology:

  • to treat the underlying disorder that caused a decrease in rhythm;
  • to correct bradycardia with medicines( xanthines, anticholinergics);
  • install a pacemaker( pacemaker) - with a high risk of cardiac arrest.

Tactics depends on the stage of bradyarrhythmia, the presence of the clinic. If the slowing of the rhythm is associated with the defeat of the myocardium - there is no complete recovery.

Physiological bradycardia in trained people and asymptomatic bradyarrhythmias do not require treatment.

Diagnosis of the heart in its work with interruptions

  1. Thorough examination of the patient to identify characteristic complaints.
  2. ECG - the "gold standard" in the installation of the type of arrhythmia. In paroxysmal forms, an episode of the disorder is not always recorded.
  3. ECG registration during the day( holter monitoring) or weeks( event monitoring) - diagnosis of arrhythmia paroxysms.
  4. ultrasound of the heart through the thorax and( or) the esophagus. To assess the function of the myocardium, the examination of the heart cavities on the subject of clots.

Prognosis for

All arrhythmias of non-cardiac origin are completely curable when correcting the original pathology.

For forms associated with myocardial damage, the prognosis depends on the degree of circulatory disturbance. Chronic cardiac pathologies require constant treatment and gradually progress.

Paroxysmal forms of heart failure are a common cause of sudden death.

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