From this article you will learn: how the heart works with ventricular tachycardia, why it arises, what symptoms are manifested, what its danger is. Types, changes in ECG, diagnostic methods and treatment regimen.
The mechanism of development of ventricular tachycardia
Ventricular tachycardia( abdominal vaginosis) is one of the most unfavorable and dangerous types of arrhythmia. This is not an independent disease, but a manifestation or consequence of a serious pathology of the heart muscle. It is characterized by the increase in the contraction of the ventricles more than 100 beats per minute. In this case, the source of electrical excitation is not the sinus node, as physiologically laid, but the focus in the ventricular muscle, which generates pathological, very often occurring electrical impulses.
Pathological impulse with ventricular tachycardia A brief summary of the facts about this pathology:
- Such a tachycardia often begins and stops suddenly.
- results in a significant disruption of the cardiac and total blood flow.
- Severity of clinical manifestations is associated with the duration and severity of ventricular tachycardia.
- Characteristic for her symptomatology is often observed by cardiologists with ischemic heart disease and myocardial infarction.
- This violation of the rhythm of the heart is extremely dangerous development of collapse - a sharp drop in blood pressure with deterioration of blood supply to vital organs - and sudden death. Therefore, it requires immediate medical assistance.
Such a rhythm disturbance does not completely cure, but it is extremely important to stop tachycardia attacks as soon as possible in order to avoid a fatal outcome from cardiac arrest.
Diagnosis and treatment of pathology is handled by a cardiologist. In specialized centers of large cities, you can turn to arrhythmology.
Mechanism of development of ventricular tachycardia
Tachycardia - an increase in the number of heartbeats more often than 80 beats per minute. Frequent pulse can be a variant of the norm under stress, physical overload, fever, running, after drinking strong coffee, etc. In these cases, electrical impulses arise in the sinus node, after which they spread from the atria to the ventricles. This process is physiologically correct. The rhythm is normalized after a short time.
Transmission of electrical impulse in a healthy heart Ventricular tachycardia is never the norm - it is a formidable signal about a problem in the heart muscle that requires emergency care. The ventricles begin to contract independently, the process of their excitation occurs in the reverse order - without impulses from the sinus node, which are blocked. Myocardial cells have a feature: unlike ordinary muscle cells, they are capable of automatism, that is, to the automatic occurrence of excitation. Thanks to this, in critical situations, the heart beats to the last, saving a person's life. With non-paroxysmal ventricular tachycardia, the frequency of contractions reaches 130, and with paroxysmal up to 220 beats per minute and more.
With too frequent heart contractions, the ventricles do not have time to fully fill up with blood. As a result, the volume of blood outflow decreases, blood pressure drops, organs and tissues are not sufficiently supplied with blood, therefore oxygen and nutrients are lost, toxins and decay products are disrupted.
The human circulatory system. Click on the photo to enlarge The abnormal foci of contractions, called ectopic in medicine, appears more often in the myocardium of the left ventricle.
Causes of development of ventricular tachycardia
The most common cause of this type of heart rhythm disturbance is the pathology of the heart. Among them:
- Acute myocardial infarction. In 90% of cases it provokes the development of ventricular tachycardia.
- Ischemic heart disease with a lack of circulation in the ischemic area of the heart muscle.
- Rheumatism or inflammatory heart disease. For example, myocarditis - inflammation of the myocardium is more often of an infectious nature.
- Different types of cardiomyopathy - damage to the heart muscle with the development of dystrophic and sclerotic processes in it.
- An aneurysm, especially in the area of the left ventricle, is the bulging of the wall of a large artery or aorta with its possible rupture.
- Congenital heart defects, for example, defects - irreversible changes in the structure of the heart.
- Right ventricular arrhythmogenic dysplasia is a progressive gradual replacement of muscle tissue by connective or fat.
Other causes:
- Surgery to the heart.
- Violation of endocrine or nervous regulation leads to a change in the cardiac tissue flow of electrical processes.
- Shift electrolyte balance - a decrease in the content of potassium, magnesium and other electrolytes involved in metabolic processes of the body.
- Overdose of certain drugs - cardiac glycosides, beta-blockers, antiarrhythmics.
- Intoxication from excessive amounts of alcohol or the use of a surrogate, as well as from narcotic substances, especially cocaine.
- Hereditary pathologies. Among them, Brugada syndrome, which is high risk of ventricular fibrillation - frequent chaotic reduction of their different sites. On the electrocardiogram there are specific changes that indicate the presence of such a genetic disease.
If the patient has no acquired heart disease and congenital anomalies of its development, but there is a ventricular tachycardia, the cause of which is not clear, then it is considered idiopathic.
Types of ventricular tachycardia
| Categories | Species with a brief definition of |
|---|---|
| By the time of occurrence of | Paroxysmal - the attack begins suddenly, heart beats reach 130 beats per minute and more. Immediate medical attention is required. Non-paroxysmal - manifested more often by group extrasystoles. Does not have a paroxysmal character. Emergency measures do not require, but with the treatment you can not pull. |
| In the form of | Monomorphic - with 1 pathological source of rhythm, in most cases is a consequence of heart damage. Polymorphic - has several foci of pulse production in the myocardium. It often develops in genetic diseases or overdose of drugs. |
| Paroxysmal resistant - characterized by the registration of several ectopic complexes on the ECG for more than 30 seconds. Pulse reaches 200 beats per minute. Significantly affects hemodynamics and increases the risk of death. Paroxysmal VT of unstable type - specific changes in the electrocardiogram are fixed for 30 seconds. Chronic - a prolonged course, sometimes several months, during which the patient repeatedly repeated relatively short bouts of ventricular tachycardia. With a continuously recurring course, circulatory disorders increase gradually. |
Symptoms of pathology
At first the disease proceeds asymptomatically. It is detected only when the patient is examined, specifically during the daily monitoring by Holter, if he has a complaint about the heart. VT in patients manifests itself in different ways, depending on the severity of the underlying heart disease.
Daily Holter monitoring The following signs indicate the onset of an attack of ventricular tachycardia:
- Increased palpitations, sensations reminiscent of frequent fluttering of the heart. The patient feels how it works.
- The appearance of a "coma" in the throat, "faintness".
- Shortness of breath.
- Compression or stitching of the sternum.
- Dizziness, right up to a presumptuous state or loss of consciousness.
- Severe weakness.
- Blurred skin, cold sweat.
- Visual impairment: doubling, blurring of objects, appearance of "flies" or "black circles" before the eyes.
- Feeling of panic and fear.
Diagnosis
Diagnosis is made by the cardiologist after a comprehensive diagnosis, including:
- Clarifying and analyzing patient complaints.
- Case history. Interrogation about the presence or absence of hereditary predisposition or existing genetic diseases, as well as the possible transfer of myocardial infarction. The doctor finds out what medicines the patient takes, etc.
- A physical examination consists of several manipulations that help the doctor objectively judge the general condition of the subject and specifically about the work of his heart. The cardiologist performs an external examination of the skin and mucous membranes, measures blood pressure, counts the number of heartbeats, listens to the heart sounds with a phonendoscope. But with VT, if there is no seizure, such a survey is of little effect. Therefore, the following studies are mandatory.
| Instrumental methods | Laboratory diagnostics |
|---|---|
| An electrocardiogram is the main method for detecting VT.It is necessary to detect abnormalities in the work of the heart, the presence of a blockade of the bundle of the Hisnus and other changes indicative of the ventricular tachycardia itself or the cause of its occurrence. | A general blood test is necessary to exclude the inflammatory process and to identify a possible cause of heart rhythm disturbances. |
| Holter monitoring of ECG is the second most important diagnostic method for determining VT.Continuous removal of the ECG during the day not only can confirm the presence of such a tachycardia, but also to understand the conditions of its appearance, how many it was, the duration of each where the focus of the rhythm is located. | Biochemical analysis - is necessary to determine the disease that led to the appearance of ventricular tachycardia. Important indicators of cholesterol, lipoproteins, triglycerides, potassium, sugar and others. |
| Echocardiography is the study of heart structures using ultrasound waves reflected from organ tissues. It is necessary to find the cause of VT.Helps evaluate the contractility of the myocardium, the state of the valvular apparatus and other cardiac structures. | |
| Ordinary computer tomography or multispiral. The difference between the latter and the first is that the emitter of electromagnetic waves is in motion, rotating in a circle along the patient's body. Identifies diseases that triggered tachycardia. | |
| Coronaroangiography is a radiopaque method for assessing the state of the heart vessels. | |
| Ventriculography is a study of the ventricles of the heart using X-rays and a contrast agent injected into a vein before the procedure. |
In addition to the above examinations in the diagnosis of VT informative load tests. One of them - veloergometry - studying the work of the heart against a background of gradually increasing load. The patient sits on a veloergometer( a special exercise bike) and twists the pedals, imitating cycling. At the same time, an electrocardiogram is recorded.
Ventricular tachycardia: changes on the ECG
Ventricular tachycardia on the ECG is manifested by certain criteria:
- Heart rate exceeds 100-140 beats per minute and can reach up to 300.
- Deviation to the left of the electrical axis of the heart( EOS).
- QRS complex expansion 0.14-0.16 sec.
- Deformation of complexes reflecting ventricular contractions. Change in their shape is observed not for all types of VT.On this basis, the doctor can determine the form of tachycardia. In the polymorphic type of VT, significant changes in QRS from the cycle to the cycle are noted on the ECG.With bi-directional tachycardia, not only this complex, but also EOS changes in each cycle.
Treatment of
Ventricular tachycardia can not be completely cured. In this case, the question is about the speedy restoration of the normal rhythm in an attack and the decrease in the frequency of their occurrence. Drug therapy is mandatory, and with paroxysmal resistant tachycardia, medical care must be provided promptly. Normalize the pulse can be an electrical impulse when conducting electropulse therapy.
The main objectives of therapy
- Treatment of the underlying disease that causes ventricular tachycardia.
- Timely and competent help in attacks of paroxysmal tachycardia for the restoration of the right heartbeats.
- Prevention of repeated episodes of VT.
Drug Therapy
- Antiarrhythmic drugs. Restore and maintain the right heart rate.
- Beta-adrenoblockers - reduce the pulse rate, reduce A / D.
- Calcium antagonists. Weaken the strength of the contractions of the heart, normalize the rhythm, dilate blood vessels, lower blood pressure.
At the onset of ventricular fibrillation, the only immediate way to restore the rhythm is to perform defibrillation with an electric charge. But you need to do this very quickly, because in a few minutes the patient can die.
Surgical treatment
- Implantation of a cardioverter-defibrillator restoring the rhythm of heart contractions. This is the best option for frequent attacks of persistent paroxysmal tachycardia.
- Radiofrequency ablation - the operation is aimed at destroying the pathological source of rhythm by the radiofrequency pulse, in those cases when its localization is definitely established.
- Implantation of an artificial pacemaker pacemaker - a small device that is sewn under the skin in the area of the left armpit, with a probe electrode located in the right ventricle or atrium. The device imposes the right rhythm on the heart and helps to keep it from it.
Implantation of the pacemaker Possible complications of the VT
- Ventricular fibrillation is a disorderly, very frequent and irregular contraction of different ventricular fibers.
- Cardiac failure is a condition that develops with prolonged fluid flow and is caused by a gradual weakening of the contractile capacity of the heart muscle.
- Pulmonary problems: shortness of breath, pulmonary edema.
- Sudden death due to cardiac arrest.
Forecast
Without treatment, the prognostic data for VT are extremely unfavorable. Patients die from ventricular fibrillation, acute failure, or cardiac arrest. Adequate, timely begun treatment significantly improves prognosis. If paroxysms in time stop, contractile function of the myocardium is not reduced, heart failure is absent, then patients live a habitual life for many years.
Therefore, at the first signs of the appearance of a frequent messy pulse, especially if the diagnosis of "ventricular tachycardia" is already established, it is vital to immediately contact the cardiologist and immediately begin the prescribed treatment.
